A Vitamin: Bad for Bones or Not? by Berkeley Wellness – A Vitamin supplements took a great success in 2002 when the famous Nurses Health Study from Harvard University linked high intakes to hip fractures in postmenopausal women. Particularly, in those consuming at least 6, 600 IU of preformed vitamin A a day from food and supplements, the potential risk of hip fracture nearly doubled. For many women, the largest source of vitamin A was multivitamins, which typically supplied 5, 000 IU back then. Since 2002, research on the effect of vitamin A on bone density and fracture risk has produced unclear results.
And some supplement companies are once more promoting formulas with high doses of vitamin A, making claims the concerns were unfounded. Beta carotene: better than A? A Vitamin is vital for health, specifically for growth, reproduction, vision, immune function and, yes, bone tissue health. You may get the vitamin two basic ways: by consuming preformed vitamin A, which comes only from animal products, nutritional supplements and fortified foods, or by consuming beta carotene from vegetables and fruits. The beta carotene is converted to vitamin A as needed in our bodies. Supplements, fortified foods and some prescription drugs contain forms of retinol like retinyl acetate, retinyl palmitate, tretinoin and retinoic acid.
The potential bone issues are posed only by preformed vitamin A, not beta carotene. High doses of vitamin A may also causes birth defects and, over the long term, serious harm to the liver, where the vitamin is stored. You cannot get toxicity levels of vitamin A by consuming beta carotene. A Vitamin deficiency is rare in the U.S., in part because milk and some other foods are fortified with it, though it’s quite common in many developing nations.
A Vitamin: inconsistent studies? Some research has supported the findings of the nursing health study, but others have found no decrease in bone density or no rise in the potential risk of osteoporotic fractures. An analysis of data from the Womens Health Initiative Observational Study in the year 2009, for example, found that high vitamin A intake wasn’t linked with fracture risk, except modestly in women with low D vitamin intake. And in 2012 an Australian study published in Osteoporosis International which tested very high doses of vitamin A as a possible cancer preventative in asbestos workers for anyplace from one to 16 years found no increase in fracture risk.
Nevertheless, a Spanish study published in the Archives of Osteoporosis this year reported that the 36 percent of postmenopausal women with the highest blood levels of vitamin A were up to eight times less unlikely to have osteoporosis than those with low levels. The risk was greatest in women who’d low blood levels of vitamin D as well as high retinol levels. Unsurprisingly, several review papers concluded that studies have been inconsistent and that more studies are needed. The inconsistencies can be due in part to the different methodologies of the studies.